Grünberg Found Useful Effect That Went Against Then-Dominant Theory

(p. A25) Peter Grünberg, a Nobel-Prize-winning physicist who discovered how to store vast amounts of data by manipulating the magnetic and electrical fields of thin layers of atoms, making possible devices like the iPad and the smartphone, has died at 78.
. . .
Since the British physicist Lord Kelvin first wrote about the subject in 1857, it had long been known that magnetic fields could affect the electrical resistance of magnetic materials like iron. Current flowed more easily along the field lines than across them.
While this effect on electrical resistance was useful for sensing magnetic fields and, in electronic heads, reading magnetic disks, it amounted to only a small change in the resistance, and physicists did not think there were many prospects for improvement.
So it was a surprise in 1988 when groups led by Dr. Fert at the Laboratoire de Physique des Solides in Paris and by Dr. Grünberg found that super-slim sandwiches of iron and chromium that they had assembled showed large sensitivity to magnetic fields — or “giant magnetoresistance,” as Dr. Fert called it. The name stuck.
The reason for the effect has to do with what physicists call the spin of electrons — their somewhat mysterious ability to have an orientation in space. When the magnetic layers of the sandwich have both their fields pointing in the same direction, electrons whose spin points along that direction can migrate freely through the sandwich. Electrons that point in another direction, however, are scattered.
If, however, one of the magnetic layers is perturbed by, say, reading a small signal, it can flip its direction so that its field runs opposite to the other one; this dramatically increases the electrical resistance of the sandwich.
As Philip Schewe, of the American Institute of Physics, explained, “You’ve leveraged a weak bit of magnetism into a robust bit of electricity.”
Experts said the discovery was one of the first triumphs of the new field of nanotechnology, the ability to build and manipulate assemblies of atoms only a nanometer (a billionth of a meter) in size.

For the full obituary, see:
DENNIS OVERBYE. “Peter Grünberg, 78, Dies; Heart of Modern Gadgets Is Based on His Research.” The New York Times (Friday, April 13, 2018): A25.
(Note: ellipsis added.)
(Note: the online version of the obituary has the date APRIL 12, 2018, and has the title “Peter Grünberg, 78, Winner of an ‘iPod Nobel,’ Is Dead.”)

Labor-Intensive Tinkering Can Advance Science

(p. A24) When John E. Sulston was 5 years old and growing up in Britain, the son of an Anglican priest, his parents sent him to a private school. There, he discovered, sports were his nemesis.
“I absolutely loathed games,” he said. “I was hopeless.”
When it came to schoolwork, he said, he was “not a books person.”
He had only one consuming interest: science. He liked to tinker, to figure out how things were put together.
. . .
The Nobel he received, shared with two other scientists, recognized the good data he amassed in his work on the tiny transparent roundworm C. elegans in an effort to better understand how organisms develop.
. . .
At the time, it was widely believed that the 558 cells the worm had when it hatched were all it would ever have. But Dr. Sulston noticed that, in fact, the worm kept gaining cells as it developed. And by tracing the patterns of divisions that gave rise to those new cells, he found, surprisingly, that the worm also lost cells in a predictable way. Certain cells were destined to die at a specific time, digesting their own DNA.
Dr. Sulston’s next major project was to trace the fate of every single cell in a worm. It was a task so demanding and labor-intensive that other scientists still shake their heads in amazement that he got it done.
Each day, bending over his microscope for eight or more hours, he would start with a worm embryo and choose one of its cells. He would then watch the cell as it divided and follow each of its progeny cells as, together, they grew and formed the organism. This went on for a total of 18 months.
In the end, he had a complete map of every one of the worm’s 959 cells (not counting sperm and egg cells).

For the full obituary, see:
GINA KOLATA. “John Sulston, 75; Tiny Worm Guided Him to Nobel.” The New York Times (Friday, March 16, 2018): A24.
(Note: ellipses added.)
(Note: the online version of the obituary has the date MARCH 15, 2018, and has the title “John E. Sulston, 75, Dies; Found Clues to Genes in a Worm.”)

“Science Didn’t Lie”

(p. 22) In the words of The Saturday Evening Post: “If America doesn’t keep out the queer, alien, mongrelized people of Southern and Eastern Europe, her crop of citizens will eventually be dwarfed and mongrelized in turn.”
According to Thomas C. Leonard, who teaches at Princeton, the driving force behind this and other such laws came from progressives in the halls of academia — people who combined “extravagant faith in science and the state with an outsized confidence in their own expertise.” “Illiberal Reformers” is the perfect title for this slim but vital account of the perils of intellectual arrogance in dealing with explosive social issues. Put simply, Leonard says, elite progressives gave respectable cover to the worst prejudices of the era — not to rabble-rouse, but because they believed them to be true. Science didn’t lie.
But barring undesirables was only half the battle; the herd also had to be culled from within. In 1907, Indiana became the first state to legalize forced sterilization, starting a landslide endorsed by progressive icons like Theodore Roosevelt and the birth control champion Margaret Sanger.

For the full review, see:
DAVID OSHINSKY. “No Justice for the Weak.” The New York Times Book Review (Sunday, March 20, 2018): 1 & 22-23.
(Note: the online version of the review has the date March 14, 2018, and has the title “‘Imbeciles’ and ‘Illiberal Reformers’.”)

The book under review, is:
Leonard, Thomas C. Illiberal Reformers: Race, Eugenics, and American Economics in the Progressive Era. Princeton, NJ: Princeton University Press, 2016.

The Role of Progressives in the Forced Sterilization of Thousands

(p. 22) Progressivism was always more than a single cause, however. Attracting reformers of all stripes, it aimed to fix the ills of society through increased government action — the “administrative state.” Progressives pushed measures ranging from immigration restriction to eugenics in a grotesque attempt to protect the nation’s gene pool by keeping the “lesser classes” from reproducing. If one part of progressivism emphasized fairness and compassion, the other reeked of bigotry and coercion.
“Imbeciles,” by Adam Cohen, the author of “Nothing to Fear: FDR’s Inner Circle and the Hundred Days That Created Modern America,” examines one of the darkest chapters of progressive reform: the case of Buck v. Bell. It’s the story of an assault upon thousands of defenseless people seen through the lens of a young woman, Carrie Buck, locked away in a Virginia state asylum. In meticulously tracing her ordeal, Cohen provides a superb history of eugenics in America, from its beginnings as an offshoot of social Darwinism — ­human survival of the fittest — to its rise as a popular movement, advocating the state-sponsored sterilization of “feeble­minded, insane, epileptic, inebriate, criminalistic and other degenerate persons.”

For the full review, see:
DAVID OSHINSKY. “No Justice for the Weak.” The New York Times Book Review (Sunday, March 20, 2018): 1 & 22-23.
(Note: the online version of the review has the date March 14, 2018, and has the title “‘Imbeciles’ and ‘Illiberal Reformers’.”)

The book under review, is:
Cohen, Adam. Imbeciles: The Supreme Court, American Eugenics, and the Sterilization of Carrie Buck. New York: Penguin Press, 2016.

Proof of Concept for Regenerating Limbs and Internal Organs

(p. D3) Scientists have decoded the genome of the axolotl, the Mexican amphibian with a Mona Lisa smile. It has 32 billion base pairs, which makes it ten times the size of the human genome, and the largest genome ever sequenced.
The axolotl, endangered in the wild, has been bred in laboratories and studied for more than 150 years. It has the remarkable capacity to regrow amputated limbs complete with bones, muscles and nerves; to heal wounds without producing scar tissue; and even to regenerate damaged internal organs.
This salamander can heal a crushed spinal cord and have it function just like it did before it was damaged. This ability, which exists to such an extent in no other animal, makes its genes of considerable interest.
. . .
The researchers have identified some of the genes involved in regeneration, and some genes that exist only in the axolotl, but there is much work still to be done.

For the full story, see:
NICHOLAS BAKALAR. “TAKE A NUMBER; 32 Billion.” The New York Times (Tuesday, February 6, 2018): D3.
(Note: ellipsis added.)
(Note: the online version of the story has the date FEB. 1, 2018, and has the title “TAKE A NUMBER; The Smiling Axolotl Hides a Secret: A Giant Genome.”)

Serial Breakthrough Innovators Have “Almost Maniacal Focus”

(p. C4) It’s 6 a.m., and I’m rushing around my apartment getting ready to fly to California to teach an innovation workshop, when my 10-year-old son looks at me with sad eyes and asks, “Why are you always busy?” My heart pounds, and that familiar knife of guilt and pain twists in my stomach. Then a thought flickers through my head: Does Jeff Bezos go through this?
I recently finished writing a book about innovators who achieved multiple breakthroughs in science and technology over the past two centuries. Of the eight individuals I wrote cases about, only one, Marie Curie, is a woman. I tried to find more, even though I knew in my scientist’s heart that deliberately looking for women would bias my selection process. But I didn’t find other women who met the criteria I had laid out at the beginning of the project.
. . .
The politically correct thing to say at this point is that expanding the roster of future innovators to include more women will require certain obvious changes in how we handle family life: Men and women should have more equal child-care responsibilities, and businesses (or governments) should make affordable, quality child care more accessible. But I don’t think it is as simple as that.
In my own case, I can afford more child care, but I don’t want to relinquish more of my caregiving to others. From the moment I first gave birth, I felt a deep, primal need to hold my children, nurture them and meet their needs. Nature is extremely clever, and she has crafted an intoxicating cocktail of oxytocin and other neurochemicals to rivet the attention of parents on their children.
The research on whether this response is stronger for mothers than for fathers is inconclusive. It is tough to compare the two, because there are strong gender differences in how hormones work. Historically, however, women have taken on a larger share of the caregiving responsibilities for children, and many (myself included) would not have it any other way.
Is such a view hopelessly retrograde, a rejection of hard-won feminist achievements? I don’t think so.
The need to connect with our children does not prevent women from being successful. There are many extremely successful women with very close relationships with their children. But it might get in the way of having the almost maniacal focus that the most famous serial breakthrough innovators exhibit.
I’m no Marie Curie, but I do have obsessive tendencies. If I did not have a family, I would routinely work until 4 a.m. if I had an interesting problem to chase down. But now I have children, and so at 5 p.m., I need to dial it back and try to refocus my attention on things like homework and making dinner. I cannot single-mindedly focus on my work; part of my mind must belong to the children.
This doesn’t mean that mothers cannot be important innovators, but it might mean that their careers play out differently. Their years of intense focus might start later, or they might ebb and surge over time. The more we can do to enable people to have nonlinear career paths, the more we will increase innovation among women–and productivity more generally.

For the full commentary, see:
Melissa Schilling. “Why Women Are Rarely Serial Innovators; A single-minded life of invention is hard to combine with family obligations. One solution: ‘nonlinear’ careers.” The Wall Street Journal (Saturday, Feb. 3, 2018): C4.
(Note: ellipsis added.)
(Note: the online version of the commentary has a date of Feb. 2, 2018.)

Schilling’s commentary is related to his book:
Schilling, Melissa A. Quirky: The Remarkable Story of the Traits, Foibles, and Genius of Breakthrough Innovators Who Changed the World. New York: PublicAffairs, 2018.

Extent of Future Global Warming Remains “Stubbornly Uncertain”

(p. A15) . . . , an exemplary French report . . . begins, “But uncertainty about how hot things will get also stems from the inability of scientists to nail down a very simple question: By how much will Earth’s average surface temperature go up if the amount of CO2 in the atmosphere is doubled?”
“That ‘known unknown’ is called equilibrium climate sensitivity (ECS), and for the last 25 years the U.N.’s Intergovernmental Panel on Climate Change (IPCC)–the ultimate authority on climate science–has settled on a range of 1.5 C to 4.5 C.”
The French report describes a new study by climate physicists Peter Cox and Mark Williamson of the University of Exeter and Chris Huntingford of the U.K.’s Center for Ecology and Hydrology. Not only does it narrow the range of expected warming to between 2.2 and 3.4 degrees Celsius, but it rules out the possibility of worrying outcomes higher than 4 degrees.
. . .
. . . , [the IPCC] backpedaled in 2013 to adopt a wider range of uncertainty, and did so entirely in the direction of less warming.
. . .
The IPCC’s new estimate was no more useful or precise than one developed in 1979 by the U.S. National Research Council, when computers and data sets were far more primitive.
This 40-year lack of progress is no less embarrassing for being thoroughly unreported in the mainstream press. The journal Nature, where the new study appears, frankly refers to an “intractable problem.” In an accompanying commentary, a climate scientist says the issue remains “stubbornly uncertain.”

For the full commentary, see:
Holman W. Jenkins, Jr. “BUSINESS WORLD; Good Climate News Isn’t Told; Reporting scientific progress would require admitting uncertainties.” The Wall Street Journal (Wednesday, Feb. 28, 2018): A15.
(Note: ellipses added.)
(Note: the online version of the commentary has the date Feb. 27, 2018.)

The “new study” in Nature, mentioned above, is:
Cox, Peter M., Chris Huntingford, and Mark S. Williamson. “Emergent Constraint on Equilibrium Climate Sensitivity from Global Temperature Variability.” Nature 553, no. 7688 (Jan. 18, 2018): 319-322.

New Technology Reveals Fossil Secrets

(p. A11) Using a new laser imaging technique to reveal traces of soft tissue in fossils of an early feathered, birdlike dinosaur, scientists have found direct evidence of a wing structure needed for flight that was previously invisible from the preserved bone evidence.
The research is part of a body of work on the cutting edge of paleontology, leveraging new technology to flesh out the study of fossils beyond bones, to look at soft tissue and feathers. Other scientists have recently turned up evidence of the protein collagen preserved in dinosaur fossils millions of years old, and scanned feathers, muscle, skin and ligament tissue from a dinosaur’s tail preserved in amber.
Known as laser-stimulated fluorescence, the new imaging technique “is revealing information preserved in the fossil we can’t see with normal light,” says University of Hong Kong paleontologist Michael Pittman, one of the leaders of the research, published Tuesday [February 28, 2017] in Nature Communications.

For the full story, see:
Ellie Kincaid. “Imaging Reveals Soft Tissue in Dinosaur Fossil.” The Wall Street Journal (Weds., March 1, 2017): A11.
(Note: bracketed date added.)
(Note: the online version of the story has the date Feb. 28, 2017, and has the title “New Imaging Method Helps Scientists Look Beyond Dinosaur Bones.”)

Innovation Skeptics Fail to See Its Broad Benefits

(p. B11) Professor Juma died on Dec. 15 [2017] at his home in Cambridge, Mass. He was 64. His wife said the cause was cancer. At his death he was widely credited as having been an important force in ensuring that biotechnology would play a critical role in improving economic life in many developing countries, especially in sub-Saharan Africa.
“Calestous understood that people often resist the changes that come with innovation, and that overcoming this resistance can be very important in enabling societies to move ahead,” said Douglas W. Elmendorf, dean of the Kennedy School. “So he tried to understand why people resist innovation, and what can be done to make them feel comfortable with change.”
Professor Juma’s latest book, “Innovation and Its Enemies” (2016), described how technological change is often greeted with public skepticism. Beneath such opposition, he argued, is the belief that only a small segment of society will benefit from potential progress, while the much broader society bears the greatest risk.
. . .
Professor Juma could be lighthearted in the classroom or in public in order to make his points. With more than 100,000 followers on Twitter, he shared with them cartoons that teased skeptics of science and innovation. One of his last posts featured a game show called “Facts Don’t Matter.” In it, a contestant is told: “I’m sorry, Jeannie, your answer was correct, but Kevin shouted his incorrect answer over yours, so he gets the points.”

For the full obituary, see:
ADEEL HASSAN. “Calestous Juma, 64, Advocate of African Progress, Dies.” The New York Times (Tues., January 2, 2018): B11.
(Note: ellipsis, and bracketed year, added.)
(Note: the online version of the obituary has the date JAN. 1, 2018, and has the title “Calestous Juma, 64, Dies; Sought Innovation in African Agriculture.”)

The most recent book by Juma, mentioned above, is:
Juma, Calestous. Innovation and Its Enemies: Why People Resist New Technologies. New York: Oxford University Press, 2016.

World War I Spread the Deadly Flu of 1918

(p. A17) The Spanish flu began in the spring of 1918, infected 500 million people, and killed between 50 million and 100 million of them–more than both world wars and the Holocaust combined. Not since the bubonic plague of the mid-14th century–the Black Death–had such a fearsome pestilence devastated mankind.
Spanish-flu patients “would soon be having trouble breathing,” writes Laura Spinney in “Pale Rider,” her gripping account of the pandemic.
. . .
Ms. Spinney is at her best in trying to tease out the real origin of the pandemic. The first suspect was China, where pneumonic plague had erupted on the Manchurian border in 1910. The government, trying to curry favor with the Allies in World War I, had then sent tens of thousands of laborers, many infected, to dig trenches on the Western Front. Another theory put the initial outbreak at the British army’s mobilization base in Étaples in northern France. A third candidate was in the American heartland, at a U.S. Army staging base, Camp Funston in Kansas. The question is unsettled, but plainly the movement of troops in the Great War accelerated the flu’s spread.
. . .
The frantic search for the cause of the pandemic was nightmarish, too. A respected researcher persuaded himself and others that he had found the bacillus, and he persisted even though autopsies rarely turned up his pet suspect in the tissues of the dead. The microbe hunters couldn’t find their quarry because it slipped through the ultrafine strainers they tried to catch it with, and it was invisible to their microscopes. It was what the French bacteriologist Émile Roux called an “être de raison,” an organism whose existence could be deduced only from its effects. Eventually a virus–1/20th the size of a bacillus–was identified as the culprit. It was not actually seen until decades later with the invention of the electron microscope.

For the full review, see:
Edward Kosner. “BOOKSHELF; A World Of Sickness; The Spanish flu of 1918-19 infected 500 million people, killing between 50 and 100 million. Its cause was discovered only decades later.” The Wall Street Journal (Monday, Dec. 11, 2017): A17.
(Note: ellipses added.)
(Note: the online version of the review has the date Dec. 10, 2017, and has the title “BOOKSHELF; Review: A World of Sickness; The Spanish flu of 1918-19 infected 500 million people, killing between 50 and 100 million. Its cause was discovered only decades later.”)

The book under review, is:
Spinney, Laura. Pale Rider: The Spanish Flu of 1918 and How It Changed the World. New York: PublicAffairs, 2017.

High Demand for STEM Workers Is Mainly High for Workers in Info Tech

(p. 10) A working grasp of the principles of science and math should be essential knowledge for all Americans, said Michael S. Teitelbaum, an expert on science education and policy. But he believes that STEM advocates, often executives and lobbyists for technology companies, do a disservice when they raise the alarm that America is facing a worrying shortfall of STEM workers, based on shortages in a relative handful of fast-growing fields like data analytics, artificial intelligence, cloud computing and computer security.
“When it gets generalized to all of STEM, it’s misleading,” said Mr. Teitelbaum, a senior research associate in the Labor and Worklife Program at Harvard Law School. “We’re misleading a lot of young people.”
Unemployment rates for STEM majors may be low, but not all of those with undergraduate degrees end up in their field of study — only 13 percent in life sciences and 17 percent in physical sciences, according to a 2013 National Science Foundation survey. Computer science is the only STEM field where more than half of graduates are employed in their field.

For the full story, see:
STEVE LOHR. “Where the STEM Jobs Are/Aren’t.” The New York Times, Education Life Section (Sun., NOV. 5, 2017): 10.
(Note: the online version of the story has the date NOV. 1, 2017, and has the title “Where the STEM Jobs Are (and Where They Aren’t).”)